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It exerts its effects through multiple mechanisms, including: VEGF receptor modulation (vascular endothelial growth factor)promoting angiogenesis and blood vessel formation Growth hormone receptor interaction facilitating tissue repair and regeneration Nitric oxide pathway activation improving blood flow and reducing oxidative stress FAK-paxillin pathway signaling crucial for cell migration and wound healing Modulation of inflammatory cytokines reducing excess inflammation while preserving healing responses These are cascading processes

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The rate of loss began tapering after week 36, consistent with the metabolic adaptation seen in all obesity pharmacotherapy trials, but the plateau in most participants wasnt reached until approximately weeks 44-48

10.1006/abbi.1993.1216 Arch