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Their mechanisms are complementary rather than competitive, addressing different aspects of neural plasticity: Dihexa promotes the structural formation of new synaptic connections, while PE-22-28 enhances neuronal excitability and drives neurogenesis

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Notably, T helper 1 (Th1) cells, natural killer (NK) cells, and CTLs release interferon-gamma (IFN-), a pro-inflammatory cytokine that is crucial to vitiligo pathogenesis and CTL-mediated melanocyte death in depigmented areas

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