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Gut microbe-derived metabolites such as ROS and lipopolysaccharides can leak into the body circulation via a bidirectional microbe-gut-brain axis communication system, and these neurotoxin releases from the gut microbiota can elicit an inflammatory response and cross the bloodbrain barrier to modulate neuronal activity, and neuroinflammation has been implicated in the pathogenesis of diabetes mellitus and its associated cognitive dysfunction (124)
These findings are relatable to clinical practice, where dosing is often adjusted based on individual needs and clinical judgement, said lead investigator Timothy Garvey, MD, professor of medicine and director of the Diabetes Research Center at the University of Alabama at Birmingham
analyses of safety endpoints were based on the safety analysis set (SAS), which included all participants exposed to at least one dose of trial product
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