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However, there are no high-quality human trials, no standardized dosing, and no long-term safety data

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N1437D HOMO mutant mice harbor higher tau phosphorylation, lipofuscin accumulation, autophagy, and lysosomal abnormality in SN TH neurons at 2627 months of age Because N1437D mutations caused behavioral and histopathological damage at older ages, we selected mice aged 26 to 27 months to explore the molecular pathological features

6,7,8,9,10,43,44,45,46 In many of these studies, a possible therapeutic window was observed upon NAMPT inhibition, since the response of the normal cells was different from that of the tumor cells

Akalestou E, Miras AD, Rutter GA, le Roux CW