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Genetic polymorphisms in NMDAR subunits, such as NR1 and NR2B, influence individual sensitivity to the therapeutic and toxic effects of ketamine ( Comorbid conditions, such as diabetes and chronic kidney disease, amplify ketamines neurotoxicity by increasing oxidative stress and impairing drug metabolism

5-Amino-1MQ inhibits NNMT, preventing new fat storage and increasing the baseline metabolic rate by raising NAD+ availability

Practical takeaway: This is in vitro data, not in vivo