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Of great interest, infusion of IGF-I was shown to elicit a significant (~40%) increase in cerebromicrovascular density in the adult mouse brain[343], via stimulation of HIF-1 and its downstream effector, VEGF

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In summary, disruptions in FA metabolism mediated by astrocyte mitochondria dysfunction may contribute to the pathogenesis and progression of PD, highlighting potential targets for therapeutic interventions

Its 50-fold MC4R-over-MC1R selectivity ensures no melanogenesis at any dose within the therapeutic range