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Studies have shown that using ferroptosis-specific inhibitors can protect against severe tissue damage in these cases [5]

Instead, pro-inflammatory cytokine production and inflammasome activation in macrophages predominantly relies on the regulation of the Nrf2 response 80 , which can be primarily activated by glutathione or thioredoxin systems, as well as to a lesser extent NOXs 81

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Its unique structure and potential involvement in important bioactive pathways make it valuable for researchers exploring tissue response, cellular communication, and systemic balance

In conclusion, our results identify CP as a potential therapeutic target to protect dopaminergic neurons by reducing neurons sensitivity to ferroptosis