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doi: 10.1210/jc.2006-0573 103

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A recent investigation revealed that the expression of VRK2 was upregulated in response to ROS signals, while the link between VRK2 and metabolic rewiring remains obscure [18].In this study, we tried to explore the effects of VRK2 on the rewiring of metabolic networks and identify metabolic vulnerabilities resulting from VRK2-driven metabolic reprogramming

The increased DHA uptake in mutant cells produced oxidative stress, increasing the level of reactive oxygen species (ROS) in cells because intracellular DHA was rapidly reduced back to vitamin C at the expense of glutathione (GSH), a master antioxidant in cells

Meanwhile, NF-B (consisting of c-Rel, RelA (p65), RelB, p50/p105, and p52/p100) together with an inhibitor of NF-B kinase (IKK) proteins regulates many physiological processes, such as inflammation, innate- and adaptive-immune responses, and cell death [94,95]