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A helpful stance is to treat promising as needs better human studies, not as proven. How GHK Cu may act It is useful to group proposed mechanisms into a few broad hypothesis buckets. These buckets are based on repeated themes in the literature rather than claims of proof
Clinical trials are advancing to understand optimal dosage, long-term effects, and potential combinations with existing pharmacological interventions
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An attractive candidate for such an activity is, for example, the modulation of the redox state of key proteins involved in DNA repair such as p53 [see 46]." [9] The ability to repair DNA damage that's already been done, rather than only prevent that damage from occurring, is rare
