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In experiments conducted on mouse models lacking the ApoE gene (ApoE(-/-)), GHRP-2 exposure over 12 weeks seemed to elevate circulating levels of IGF-I, with increases observed to be between 1.2 to 1.6 times the initial measurements

PMID: 12097260

The mechanisms include reduction of excitotoxicity and oxidative stress, enhanced neurotrophic factor expression supporting neuronal survival, angiogenesis improving blood flow, and modulation of dopamine and serotonin systems

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