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Overexpression of METTL3 in lung cancer cells inhibits radiation-induced bystander effect

Proliferative Neural Stem Cells Have High Endogenous ROS Levels That Regulate Self-Renewal and Neurogenesis in a PI3K/Akt-Dependant Manner

It has been found that CL is transported to the OMM and is capable of interacting with the N-terminal structural domain of LC3, which ultimately leads to the formation of autophagosomes and promotes the onset of mitophagy in neuronal cells to remove damaged mitochondria [76]

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