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Further alterations in intracellular redox balance driven by lower GSH availability induce cell death and enhance T2 signature protein expression We previously demonstrated that 15LO1-PEBP1 complex activation consistently upregulates the expression of T2-signature gene/protein expression, including MUC5AC, periostin (POSTN), and CCL26 (6, 10, 14)

A possible reason for this is that there is a higher background concentration of carnitine in muscle than in the liver, or that the degree of supplementation with l- and d-carnitine was insufficient to significantly alter the concentration of acyl-carnitine, such that any change in lipid metabolism in muscle was not obvious