Zhao, L., Mechanism of Natural Drugs on Nonalcoholic Fatty Liver Disease
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Non-genotoxic p53 activation and blockade of Bcl-2 signaling synergistically induce apoptosis in AML through induction of Noxa and Bak/Bax heterodimerization
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Immune metabolites [13], such as serine [14], succinate [15], -ketoglutarate [16], L-carnitine (LC) [17] and fatty acids (FAs) [18], are capable to manipulate polarization by remodeling intracellular metabolism in macrophages
