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Furthermore, GLP-1 and glucose-dependent insulinotropic polypeptides have been shown to induce substance P release from sensory nerves expressing TRPV1 and TRPA1, broadening the potential pain modulation mechanisms of GLP-1 and suggesting their application in peripheral pain management 64

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Summary Preclinical studies indicate that reduced NPY signaling, especially in the CeA, may increase susceptibility for developing AUD

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[37] However, single daily dosing is sufficient to treat most cases of adult-onset GH insufficiency