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The N-terminal half of the HIV Tat protein also exploits the latter interface region when binding to cyclin T within P-TEFb (PDB ID 3MIA) 51 , thereby activating CDK9, changing its substrate specificity, and recruiting it to a sequence element within the HIV mRNA to promote transcription 51

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Oral Surg Oral Med Oral Pathol Oral RadiolEndod 1998 Feb;85(2):135-41

Consult with a qualified healthcare professional or registered dietitian about your specific nutritional requirements

Studies have examined its interaction with growth factor pathways, particularly the FAK-paxillin signaling cascade, which plays a role in cell migration and tissue remodeling