This signaling can stimulate GLP-1 release through mechanisms that include modulation of intracellular calcium and cyclic adenosine monophosphate (cAMP) signaling, as well as indirect gut, brain metabolic pathways described in experimental models
Originally published Aug 16, 2005
Once released, the free calcium interacts with a variety of proteins activating a series of biochemical reactions specific to the particular cell type and the signal initiating the calcium release
To view a copy of this licence, visit About this article Cite this article Bramble, M.S., Fourcassi, V., Vashist, N
Down-regulation of antioxidative capacity in a transgenic tobacco which fails to develop acquired resistance to necrotization caused by tobacco mosaic virus