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Key Points The majority of the genes that associate with hyperuricaemia and gout in genome-wide association studies have been implicated in the renal urate-transport system Genetic variation explains only a modest level of variance in serum uric acid levels (6%) Serum uric acid levels are determined by the net balance between urate absorption and secretion, which is mediated by separate sets of transporters in the renal proximal tubule The clinical utility of testing for urate-associated genes seems limited because serum urate levels themselves can effectively predict gout risk at a low cost Urate-associated genes and genetically determined urate levels have been largely unassociated with cardiovascular or metabolic outcomes, suggesting that serum uric acid does not have a causal role in these outcomes Strong pharmacogenetic associations between HLA-B * 5801 alleles and severe allopurinol-hypersensitivity reactions have been shown in Asian and European populations, suggesting clinical utility of testing for these alleles This is a preview of subscription content, access via your institution Access options Subscribe to this journal Receive 12 print issues and online access $189.00 per year only $15.75 per issue Buy this article Purchase on SpringerLink Instant access to the full article PDF

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Shane told Doate the bill as written relies on doctors using best practices like we do for a lot of situations, but he added that lawmakers could consider adding specific criteria or referencing established clinical guidelines into the bill language

The genetic variants analyzedGLP1R, GIPR, FTO, and MC4Rinfluence how the body responds to GLP-1 signaling and metabolic regulation