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Substituting the C-terminal tail of GLP-1R with that of GIPR resulted in a small but significant increase in the extent ( E MAX ), but not potency, to which GLP-1 stimulated Venus-mG s recruitment to this receptor (Figure 2A

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[16] [17] Early appetite changes represent the initial pharmacological effect, while measurable weight reduction reflects the cumulative impact of sustained caloric deficit over time

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