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In addition, an increase in acidic vesicular organelles (AVOs) and in LC3 II protein expression produced by NaDOC means that autophagy might be another mechanism triggered by this bile salt associated with the inhibition of the intestinal Ca 2+ absorption[22] (Table 1)

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GHK is a high-affinity copper-binding peptide that has been proposed to function as a physiological copper carrier and delivery agent moving copper between proteins and binding sites in plasma and in tissues, with downstream consequences for processes that depend on copper availability and on GHK-dependent signaling