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Tendon injuries initially suffer from: Poor blood supply Limited vascularity Slow nutrient delivery In rodent models, BPC-157 appears to: Upregulate VEGFR2 signalling Increase capillary density Improve microvascular perfusion Mechanistically: BPC-157 VEGFR2 activation PI3K/Akt pathway eNOS phosphorylation Increased nitric oxide Improved angiogenesis More vascularity = better nutrient and oxygen delivery to fibroblasts

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The study looked at those who quit within less than three months, and those who discontinued between the three-month mark and one year

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Furthermore, an analysis was performed using a patient cohort demographic profile reflecting the characteristics representative of patients with obesity in Spain, validated by clinical experts (Table S22)