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Targeting or combined with ferroptosis pathway to regulate the cerebral environment is a very advisable treatment at present, and the inhibition of ferroptosis is likely to provide a new strategy for the prevention of neuron death induced by IS

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Ros generation and autophagosome accumulation contribute to the dmamcl-induced inhibition of glioma cell proliferation by regulating the ros/mapk signaling pathway and suppressing the akt/mtor signaling pathway

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