Loren Pickart, who isolated GHK-CU in 1973, shows that the peptide increases production of Type I and Type III collagen while simultaneously upregulating matrix metalloproteinases, the enzymes that break down old, damaged collagen
The reduced form of L-glutathione is an antioxidant that protects cells and tissues by scavenging free radicals
5-ALA cellular energy boost 5-aminolevulinic acid (5-ALA) is a precursor in the heme biosynthesis pathway
Protection from DHT Damage While GHK-Cu is not a DHT (dihydrotestosterone) blocker in itself, studies indicate that it may reduce the sensitivity of hair follicles to DHT , the hormone primarily responsible for androgenetic alopecia (pattern baldness)
Key mechanisms of action include: Fibroblast activation Suppression of collagen-degrading enzymes Reduced inflammatory cytokines Enhanced granulation tissue formation Improved angiogenesis In ischemic wound models, injectable GHK-Cu restored fibroblast function and significantly accelerated skin repair compared to controls