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Remarkably, the heterologous expression of an engineered bifunctional bacterial GSH biosynthetic enzyme ( GshF ) in mitochondria enabled mitochondrial GSH production and ameliorated the metabolic and proliferative defects caused by its depletion
Prompt treatment is essential to prevent long-term neurological consequences
Indeed, research attention in the last decade has focused significantly on antioxidants targeting the mitochondria, and it appears that the focus is shifting from more traditional antioxidants (see Figure 2)
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