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The mechanism is through a variety of autophagy-related proteins (including Beclin1 and LC3B, which are the initial protein and structural components of autophagosomes, respectively) to form double membrane vesicles to engulf damaged proteins or organelles and bind to lysosomes [31]

J Infect Dis 128:1522

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Any compound that may affect inflammation or blood vessel signaling could, in theory, interact with existing conditions or medications