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We propose that hepatic activation of TH signaling has insulinotropic and glucose-lowering effects, as it is able to increase non-12-OH FXR-antagonistic BA levels through shaping the BA composition by repressing hepatic CYP8B1, thereby enhancing the GLP-1 production via suppressing intestinal FXR signaling

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Jia G, Whaley-Connell A, Sowers JR

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This intravenous therapy allows for direct and efficient delivery of this essential antioxidant into your bloodstream, bypassing the digestive system for maximum absorption