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After coronary artery ligation in mice, intraperitoneal T4 treatment upregulated cardiac ILK and Akt activity, enhanced early myocyte survival, and improved cardiac function establishing the mechanistic basis for the cardiac and broader tissue-repair claims associated with thymosin beta-4 and its 1723 fragment TB-500

( 9 , 10 ) Basically what this means is that its a better dietary/therapeutic decision to provide the orally active and absorbed S-Acetyl glutathione, which increases intracellular rGSH directly and naturally without all the increased energy expenditure and without being compromised from disease states

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Guo, Y.F

Put simply, most diseases and illnesses do not occur without something (often that we can control) causing the body to become less resilient and more susceptible to that illness or disease