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Given the potential impact of these factors on the quantification of GSH, here we examined if the type of anticoagulant used during sample collection (heparin or EDTA), deproteinization of plasma prior to storage (yes or no), storage temperature (80 o C or 4 o C), and processing time (duration of time between collection of whole blood and deproteinization of plasma) impact levels of free GSH and GSSG, in plasma

Whether the p53/p21 axis was involved in keloid immunosurveillance requires further analysis

Mechanistically, aberrant copper homeostasis during I/R impairs Fe-S cluster integrity, increases ROS production, and depletes GSH, culminating in synergistic proteotoxic and oxidative damage [160]

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