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Second, we excluded patients who had any TZD records during follow-up from the cohort entry date (i.e., GLA initiation) to the index date (i.e., liver event occurrence), given its potential liver benefit

GCLC knockdown in rat astrocytes resulted in increased oxidative stress markers, mitochondrial depolarization, and apoptotic susceptibility, whereas GCLC overexpression restored mitochondrial membrane potential and reduced apoptotic rates

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