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after stimulation of SMase-dependent ceramide formation, assembly of the death-inducing signaling complex (DISC) and oligomerization of Fas receptor, as well as cleaved caspase-8 and caspase-3, were detected [130]

Complementing these effects, KPV exerts tissue-selective anti-inflammatory activity by inhibiting NF-B signaling, reducing adhesion molecule expression, and suppressing pro-inflammatory cytokines, thereby maintaining a controlled immune environment conducive to repair

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