In response to stress, an intricate molecular mechanism facilitated by sensor cysteines within Keap1 allows Nrf2 to escape ubiquitination, accumulate within the cell, and translocate to the nucleus, where it can promote its antioxidant transcription program
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In addition, with its origins in -MSH, KPV has demonstrated therapeutic value related to immune function, namely antimicrobial and wound-healing potential
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