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This can be due to COMT, MTR or MTRR genetic mutations which help the body convert B12 into its active forms for use at the enzymatic and cellular level

How it works: TB-500 is a synthetic fragment of Thymosin Beta-4, a protein your body uses to move healing cells to damaged areas and build new blood vessels

Unfortunately, the number of people who are B12-deficient seems to be growing

The documentation must mention the source of blood loss