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Although -oxidation is impaired in the setting of VPA toxicity, this metabolite has a prolonged elimination half-life and could be responsible for the prolonged coma that is sometimes observed despite the normalization in plasma VPA concentrations [17, 18]

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References Aarsland D et al (2021) Parkinson disease-associated cognitive impairment

It confirms that the molecule reaches the receptor, activates it, and produces the expected downstream endocrine consequence a necessary condition for any hoped-for benefit