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Human thyroid C-cells express far lower GLP-1R density, and no excess medullary thyroid carcinoma signal has emerged in SURPASS/SURMOUNT human follow-up data to date, though post-marketing surveillance continues [33]

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These IPGs serve as second messengers, facilitating downstream activation of IRS, PI3K/Akt pathways, and ultimately promoting glucose uptake and metabolism (135)

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