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Via the incretin pathway, they stimulate insulin release, suppress glucagon release, slow down gastric emptying, and enhance central satiety signal

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Transitions from desensitized to resting/closed state require substantial conformational changes in LBD and the LBD-TM linkers In the gating cycle, post-desensitization, receptor needs to recover to a resting state, where the LBDs couple as 2-dimers for the next cycle of activation