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Tomita H, Tanaka K, Tanaka T, Hara A

Further encouraged by the parallel between COVID-19 and CF in terms of activation of both ENaC and TNF/NFB signaling, we have hypothesized that inflammation in the COVID-19 airway might be due to inhibition of CFTR signaling in normal lung epithelial cells by the SARS-CoV-2 spike protein

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These factors cooperate to restore the metabolic and synthetic functions accurately and timely after liver injury or resection

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