Key pathophysiological changes in acute lung injury caused by sepsis include infiltration of inflammatory cells, release of inflammatory mediators, thrombus formation in pulmonary capillaries, interstitial pulmonary edema and pulmonary fibrosis, with clinical manifestations primarily being progressive dyspnea and persistent hypoxemia secondary to sepsis (99)
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By contrast, increases in blood levels of glucose were similar between the remdesivir-treated and placebo-treated groups in two RCTs with multiethnic groups 169 and Chinese patients 192
