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Rat embryos cultured under copper-deficient conditions develop abnormally and are characterized by an impaired oxidant defense system

"Role of factor XIII in fibrin clot formation and effects of genetic polymorphisms"

These mechanisms encompass hormonal signaling interference, metabolic pathway disruption, oxidative stress induction, and epigenetic modificationseach contributing to impaired spermatogenesis, compromised sperm function, and broader endocrine dysfunction

In Perseus [29], the table with protein LFQ intensities was as well filtered for contaminants, after which the matrix was further filtered to contain at least three valid values in at least one of the groups

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