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Elevated intracellular iron levels facilitate the generation of ROS through Fenton chemistry, subsequently initiating lipid peroxidation reactions ( Compared with other forms of cell death such as apoptosis, necrosis, and autophagy, ferroptosis exhibits distinct molecular mechanisms and biological features ( Table 1 ): (1) Iron Dependence: Ferroptosis is uniquely triggered by elevated intracellular iron levels, differentiating it from other cell death modes

Indeed, given that colorectal cancer cell lines display increased expression of H 2 S-synthesising enzymes, increased expression of SOP enzymes affords a higher capacity of these cells to dispose of excess H 2 S while stimulating cell bioenergetics

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