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In lab-dish experiments, the team exposed gum cells called keratinocytes to periodontitis-associated bacteria, which induced a senescence-like molecular signature

IgM anti-ACE2 autoantibodies in severe COVID-19 activate complement and perturb vascular endothelial function

PARP1 handles about 90% of all PARP activity when DNA gets damaged

These results, together with a series of complementary experiments, confirmed that the non-canonical activity of GCLC protects cystine-starved cells from ferroptosis in a GSH-independent manner 12

Under reducing conditions sustained by GSH, TXN remains reduced and prevents NLRP3 assembly